Guel, IbrahimKarapinar, HekimYarlioglues, MikailOzdogru, IbrahimKaya, Mehmet GungorYilmaz, AhmetTurgut, Okan OnurTandogan, IzzetEryol, Namik Kemal2019-07-272019-07-282019-07-272019-07-2820110003-3197https://dx.doi.org/10.1177/0003319710377077https://hdl.handle.net/20.500.12418/9573Impairment of endothelial functions has been shown to occur after acute and chronic exposure to passive smoking (PS), as assessed by flow-mediated dilatation (FMD) of the brachial artery. A total of 61 participants, 30 male and 31 female, mean ages 26 (18-36) were enrolled in the study. All were clinically well and nonsmokers. All participants stayed for 30 minutes in the smoking room. Carbon monoxide (CO) level was 7.42 +/- 0.98 ppm (4.71-10.50). Mean carboxyhemoglobin (COHb) levels of participants were significantly elevated after PS. Mean FMD was 18.6% +/- 9% and decreased to 12.4% +/- 7% after PS (P < .001). In the current study, with more number of participants at lower CO concentrations (7.42 ppm) and with smaller increase in COHb (51%) significant reduction (33%, P < .001) in FMD was observed.en10.1177/0003319710377077info:eu-repo/semantics/closedAccessendothelial dysfunctionpassive smokingArticle623247245206826102-s2.0-79952783473Q2WOS:000288462900006Q3