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Nitric oxide synthase inhibition attenuates vasoactive response to spinal cord stimulation in an experimental cerebral vasospasm model
(SPRINGER-VERLAG WIEN, 2001)
Background. The basic mechanism of delayed cerebral vasospasm following subarachnoid haemorrhage (SAH) has been intensively investigated. II is thought that nitric oxide (NO) is a basic mediator of the cerebral vasodilator ...