Subclinical atherosclerosis in fibromyalgia syndrome

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dc.authoridYILDIZ, BULENT/0000-0003-3836-3184
dc.authoridAtik, Irfan/0000-0002-9026-2076
dc.authoridGul, Enes/0000-0002-5562-2697
dc.contributor.authorGul, Enes
dc.contributor.authorYildiz, Bulent
dc.contributor.authorAtik, Irfan
dc.contributor.authorSahin, Ozlem
dc.date.accessioned2024-10-26T18:02:21Z
dc.date.available2024-10-26T18:02:21Z
dc.date.issued2024
dc.departmentSivas Cumhuriyet Üniversitesi
dc.description.abstractIntroduction: Fibromyalgia syndrome (FMS) is a chronic disease of unknown pathophysiology in which inflammatory markers are not increased. The risk of cardiovascular disease is known to be raised in FMS. Determination of carotid intima-media thickness (CIMT) is essential for the early detection of cardiovascular disease risk. In this study, we aimed to investigate how FMS affects CIMT and its relationship with disease duration.Methods: This study was planned prospectively and included 20 female FMS patients and 20 healthy female controls. Detailed medical histories were obtained. Persons with a known chronic disease (diabetes mellitus, hypertension, thyroid function disorder, rheumatic disease, etc.), known atherosclerotic condition, body mass index >30 and smokers were excluded. Laboratory values of FMS patients and the control group in the last 3 months were recorded. The patients' and control groups' right and left common carotid arteries were imaged with ultrasonography.Results: There was no significant difference in laboratory values (although creatinine was different, it was within the normal range in both groups). The mean right common carotid artery intima-media thickness was 0.5 (+/- 0.07) mm in the patient group and 0.43 (+/- 0.05) mm in the control group (p = .005). The mean left common carotid artery intima-media thickness was 0.5 (+/- 0.06) mm in the patient group and 0.43 (+/- 0.05) mm in the control group (p = .001). Carotid intima-media thickness values were significantly higher in the patient group than in the control group.Conclusions: Although FMS is not an inflammatory disease, it causes endothelial dysfunction and atherosclerosis.
dc.description.sponsorshipSivas Cumhuriyet University Scientific Research Automation [T-880]
dc.description.sponsorshipSivas Cumhuriyet University Scientific Research Automation, Grant/Award Number: T-880
dc.identifier.doi10.1002/sono.12386
dc.identifier.endpage22
dc.identifier.issn2202-8323
dc.identifier.issn2054-6750
dc.identifier.issue1
dc.identifier.scopus2-s2.0-85177422152
dc.identifier.scopusqualityN/A
dc.identifier.startpage15
dc.identifier.urihttps://doi.org/10.1002/sono.12386
dc.identifier.urihttps://hdl.handle.net/20.500.12418/28073
dc.identifier.volume11
dc.identifier.wosWOS:001107385300001
dc.identifier.wosqualityN/A
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.language.isoen
dc.publisherWiley
dc.relation.ispartofSonography
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanı
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectcarotid intima-media thickness
dc.subjectcarotid ultrasonography
dc.subjectfibromyalgia syndrome
dc.subjectinflammation
dc.subjectsubclinical atherosclerosis
dc.titleSubclinical atherosclerosis in fibromyalgia syndrome
dc.typeArticle

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